Browsing by Author "Normandin, Marc"

Now showing items 1-4 of 4

    • Elevated Brain Cannabinoid CB1 Receptor Availability in Posttraumatic Stress Disorder: A Positron Emission Tomography Study 

      Neumeister, Alexander; Normandin, Marc D.; Pietrzak, Robert H.; Piomelli, Daniele; Zheng, Ming-Qiang; Gujarro-Anton, Ana; Potenza, Marc N.; Bailey, Christopher R.; Lin, Shu-fei; Najafzadeh, Soheila; Ropchan, Jim; Henry, Shannan; Corsi-Travali, Stefani; Carson, Richard E.; Huang, Yiyun (2013)
      Endocannabinoids and their attending cannabinoid type 1 receptor (CB1) have been implicated in animal models of posttraumatic stress disorder (PTSD). However, their specific role has not been studied in people with PTSD. ...

    • In Vivo Quantification of Mitochondrial Membrane Potential 

      Alpert, Nathaniel; Pelletier-Galarneau, Matthieu; Petibon, Yoann; Normandin, Marc; El Fakhri, Georges (Springer Science and Business Media LLC, 2020-07-08)
      Momcilovic et al1 report mitochondrial metabolism differences amongst various mouse lung cancer subtypes, as measured by positron emission tomography (PET) and a voltage-sensitive tracer. They describe their experiments ...

    • Neurovascular coupling to D2/D3 dopamine receptor occupancy using simultaneous PET/functional MRI 

      Sander, C. Y.; Hooker, Jacob M; Catana, Ciprian; Normandin, Marc D; Alpert, Nathaniel Moritz; Knudsen, G. M.; Vanduffel, Wim J.M.; Rosen, Bruce; Mandeville, Joseph B. (Proceedings of the National Academy of Sciences, 2013)
      This study employed simultaneous neuroimaging with positron emission tomography (PET) and functional magnetic resonance imaging (fMRI) to demonstrate the relationship between changes in receptor occupancy measured by PET ...

    • A Receptor-Based Model for Dopamine-Induced fMRI Signal 

      Mandeville, Joseph B.; Sander, Christin Y.M.; Jenkins, Bruce Gorton; Hooker, Jacob M; Catana, Ciprian; Vanduffel, Wim J.M.; Alpert, Nathaniel Moritz; Rosen, Bruce; Normandin, Marc D (Elsevier BV, 2013)
      This report describes a multi-receptor physiological model of the fMRI temporal response and signal magnitude evoked by drugs that elevate synaptic dopamine in basal ganglia. The model is formulated as a summation of ...