Now showing items 1-4 of 4

    • Deformed Microcavity Quantum Cascade Lasers with Directional Emission 

      Wang, Qi; Yan, Changling; Diehl, Laurent; Hentschel, Martina; Wiersig, Jan; Yu, Nanfang; Pflügl, Christian; Edamura, Tadataka; Yamanishi, Masamichi; Kan, Hirofumi; Belkin, Mikhail A.; Capasso, Federico (Institute of Physics, 2009)
      We report the experimental realization of deformed microcavity quantum cascade lasers (QCLs) with a Limaçon-shaped chaotic resonator. Directional light emission with a beam divergence of \(\theta_{\|} \approx 33^{\circ}\) ...
    • Detection and Identification of NAP-2 as a Biomarker in Hepatitis B-Related Hepatocellular Carcinoma by Proteomic Approach 

      He, Min; Qin, Jian; Zhai, Rihong; Wei, Xiao; Wang, Qi; Rong, Minhua; Jiang, Zhihua; Huang, Yuanjiao; Zhang, Zhiyong (BioMed Central, 2008)
      Background: A lack of sensitive and specific biomarkers is a major reason for the high rate of Primary hepatocellular carcinoma (HCC)-related mortality. The aim of this study was to investigate potential proteomic biomarkers ...
    • Differential Sensitivity of Melanoma Cell Lines with BRAFV600E Mutation to the Specific Raf Inhibitor PLX4032 

      Søndergaard, Jonas N; Nazarian, Ramin; Guo, Deliang; Hsueh, Teli; Mok, Stephen; Sazegar, Hooman; Barretina, Jordi G; Kehoe, Sarah M; Attar, Narsis; von Euw, Erika; Zuckerman, Jonathan E; Chmielowski, Bartosz; Comin-Anduix, Begoña; Koya, Richard C; Mischel, Paul S; Lo, Roger S; Ribas, Antoni; Wang, Qi; MacConaill, Laura Eleanor (BioMed Central, 2010)
      Blocking oncogenic signaling induced by the BRAFV600E mutation is a promising approach for melanoma treatment. We tested the anti-tumor effects of a specific inhibitor of Raf protein kinases, PLX4032/RG7204, in melanoma ...
    • Protein Aggregation and Protein Instability Govern Familial Amyotrophic Lateral Sclerosis Patient Survival 

      Agar, Jeffrey N.; Wang, Qi; Johnson, Joshua L.; Agar, Nathalie Marie Yvonne Rachel (Public Library of Science, 2008)
      The nature of the “toxic gain of function” that results from amyotrophic lateral sclerosis (ALS)-, Parkinson-, and Alzheimer-related mutations is a matter of debate. As a result no adequate model of any neurodegenerative ...