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dc.contributor.authorKometani, Kohei
dc.contributor.authorYamada, Takayuki
dc.contributor.authorSasaki, Yoshiteru
dc.contributor.authorYokosuka, Tadashi
dc.contributor.authorSaito, Takashi
dc.contributor.authorRajewsky, Klaus
dc.contributor.authorIshiai, Masamichi
dc.contributor.authorHikida, Masaki
dc.contributor.authorKurosaki, Tomohiro
dc.date.accessioned2013-10-18T19:37:40Z
dc.date.issued2011
dc.identifier.citationKometani, Kohei, Takayuki Yamada, Yoshiteru Sasaki, Tadashi Yokosuka, Takashi Saito, Klaus Rajewsky, Masamichi Ishiai, Masaki Hikida, and Tomohiro Kurosaki. 2011. CIN85 drives B cell responses by linking BCR signals to the canonical NF-κB pathway. The Journal of Experimental Medicine 208(7): 1447-1457.en_US
dc.identifier.issn0022-1007en_US
dc.identifier.urihttp://nrs.harvard.edu/urn-3:HUL.InstRepos:11189262
dc.description.abstractCIN85 transduces B cell receptor signals to IKK-β, and its expression in B cells is essential for T cell–independent type II antibody responses in mice.en_US
dc.language.isoen_USen_US
dc.publisherThe Rockefeller University Pressen_US
dc.relation.isversionofdoi:10.1084/jem.20102665en_US
dc.relation.hasversionhttp://www.ncbi.nlm.nih.gov/pmc/articles/PMC3135365/pdf/en_US
dash.licenseLAA
dc.titleCIN85 drives B cell responses by linking BCR signals to the canonical NF-κB pathwayen_US
dc.typeJournal Articleen_US
dc.relation.journalThe Journal of Experimental Medicineen_US
dash.depositing.authorRajewsky, Klaus
dc.date.available2013-10-18T19:37:40Z
dc.identifier.doi10.1084/jem.20102665*
dash.contributor.affiliatedRajewsky, Klaus


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