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Remote control of macrophage production by cancer

dc.contributor.authorCortez-Retamozo, Virnaen_US
dc.contributor.authorEngblom, Camillaen_US
dc.contributor.authorPittet, Mikael J.en_US
dc.date.accessioned2014-02-13T19:00:52Z
dc.date.issued2013en_US
dc.identifier.citationCortez-Retamozo, Virna, Camilla Engblom, and Mikael J. Pittet. 2013. “Remote control of macrophage production by cancer.” Oncoimmunology 2 (5): e24183. doi:10.4161/onci.24183. http://dx.doi.org/10.4161/onci.24183.en
dc.identifier.issn2162-4011en
dc.identifier.urihttp://nrs.harvard.edu/urn-3:HUL.InstRepos:11708548
dc.description.abstractThe communication between tumor and host cells involves signals that act across extended distances in the body. Recent evidence indicates that the hormone angiotensin II is overproduced by lung adenocarcinoma to remotely expand bone marrow-derived hematopoietic stem cells. This process amplifies the supply of tumor-associated macrophages, which promote disease progression.en
dc.language.isoen_USen
dc.publisherLandes Bioscienceen
dc.relation.isversionofdoi:10.4161/onci.24183en
dc.relation.hasversionhttp://www.ncbi.nlm.nih.gov/pmc/articles/PMC3667905/pdf/en
dash.licenseLAAen_US
dc.subjectAngiotensin IIen
dc.subjectNSCLCen
dc.subjectcanceren
dc.subjecthematopoietic stem cellen
dc.subjectlung adenocarcinomaen
dc.subjectmacrophageen
dc.subjectmonocyteen
dc.titleRemote control of macrophage production by canceren
dc.typeJournal Articleen_US
dc.description.versionVersion of Recorden
dc.relation.journalOncoimmunologyen
dash.depositing.authorEngblom, Camillaen_US
dc.date.available2014-02-13T19:00:52Z
dc.identifier.doi10.4161/onci.24183*
dash.contributor.affiliatedEngblom, Camilla
dash.contributor.affiliatedPittet, Mikael


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