Kawasaki disease patients homozygous for the rs12252-C variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions

View/ Open
Author
Bowles, Neil E
Arrington, Cammon B
Hirono, Keiichi
Nakamura, Tsuneyuki
Wee, Yin Shen
Ichida, Fukiko
Weis, John H
Published Version
https://doi.org/10.1002/mgg3.79Metadata
Show full item recordCitation
Bowles, Neil E, Cammon B Arrington, Keiichi Hirono, Tsuneyuki Nakamura, Long Ngo, Yin Shen Wee, Fukiko Ichida, and John H Weis. 2014. “Kawasaki disease patients homozygous for the rs12252-C variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions.” Molecular Genetics & Genomic Medicine 2 (4): 356-361. doi:10.1002/mgg3.79. http://dx.doi.org/10.1002/mgg3.79.Other Sources
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4113277/pdf/Terms of Use
This article is made available under the terms and conditions applicable to Other Posted Material, as set forth at http://nrs.harvard.edu/urn-3:HUL.InstRepos:dash.current.terms-of-use#LAACitable link to this page
http://nrs.harvard.edu/urn-3:HUL.InstRepos:12717563
Collections
- HMS Scholarly Articles [17875]
Contact administrator regarding this item (to report mistakes or request changes)