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dc.contributor.authorChen, Shih-Heng
dc.contributor.authorPearson, Angela
dc.contributor.authorCoen, Donald M.
dc.contributor.authorChen, Shun-Hua
dc.date.accessioned2019-10-05T03:28:14Z
dc.date.issued2004
dc.identifier.citationChen, S.-H., A. Pearson, D. M. Coen, and S.-H. Chen. 2003. “Failure of Thymidine Kinase-Negative Herpes Simplex Virus To Reactivate from Latency Following Efficient Establishment.” Journal of Virology 78 (1): 520–23. https://doi.org/10.1128/jvi.78.1.520-523.2004.
dc.identifier.issn0022-538X
dc.identifier.issn1070-6321
dc.identifier.issn1098-5514
dc.identifier.urihttp://nrs.harvard.edu/urn-3:HUL.InstRepos:41482989*
dc.description.abstractThymidine kinase-negative mutants of herpes simplex virus did not reactivate from latency in mouse trigeminal ganglia, even when their latent viral loads were comparable to those that permitted reactivation by wild-type virus. Thus, reduced establishment of latency does not suffice to account for the failure to reactivate.
dc.language.isoen_US
dc.publisherAmerican Society for Microbiology
dash.licenseLAA
dc.titleFailure of Thymidine Kinase-Negative Herpes Simplex Virus to Reactivate from Latency following Efficient Establishment
dc.typeJournal Article
dc.description.versionVersion of Record
dc.relation.journalJournal of Virology
dash.depositing.authorCoen, Donald Mark::f1d1eb8434c5ee0d3e2fa13c1a313e4d::600
dc.date.available2019-10-05T03:28:14Z
dash.workflow.comments1Science Serial ID 62863
dc.identifier.doi10.1128/JVI.78.1.520-523.2004
dash.source.volume78;1
dash.source.page520


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