Bowles, Neil EArrington, Cammon BHirono, KeiichiNakamura, TsuneyukiNgo, LongWee, Yin ShenIchida, FukikoWeis, John H2014-08-132014Bowles, Neil E, Cammon B Arrington, Keiichi Hirono, Tsuneyuki Nakamura, Long Ngo, Yin Shen Wee, Fukiko Ichida, and John H Weis. 2014. “Kawasaki disease patients homozygous for the rs12252-C variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions.” Molecular Genetics & Genomic Medicine 2 (4): 356-361. doi:10.1002/mgg3.79. http://dx.doi.org/10.1002/mgg3.79.2324-9269http://nrs.harvard.edu/urn-3:HUL.InstRepos:12717563en-USKawasaki disease patients homozygous for the rs12252-C variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesionsJournal Article2014-08-1310.1002/mgg3.79