Tolchin, BenjaminFantaneanu, TadeauMiller, MichaelHelgager, JeffreyLee, Jong2016-08-092016Tolchin, Benjamin, Tadeau Fantaneanu, Michael Miller, Jeffrey Helgager, and Jong Woo Lee. 2016. “Status epilepticus caused by cerebral amyloid angiopathy-related inflammation.” Epilepsy & Behavior Case Reports 6 (1): 19-22. doi:10.1016/j.ebcr.2016.05.003. http://dx.doi.org/10.1016/j.ebcr.2016.05.003.2213-3232http://nrs.harvard.edu/urn-3:HUL.InstRepos:27822327This report discusses a case of nonconvulsive status epilepticus, caused by cerebral amyloid angiopathy-related inflammation. Brain biopsy demonstrated cerebral amyloid angiopathy, with clinical and radiographic features indicative of a fluctuating inflammatory process. Immunomodulatory treatment with pulse steroids resulted in rapid and dramatic clinical and radiographic improvement. Cerebral amyloid angiopathy-related inflammation should be considered in the differential diagnosis of new-onset seizures after the age of 40, when associated with fluctuating multifocal T2 hyperintensities and petechial hemorrhages on gradient echo (GRE) or susceptibility-weighted (SWI) MRI sequences.en-USCerebral amyloid angiopathyInflammationStatus epilepticusMRIEEGPathologyStatus epilepticus caused by cerebral amyloid angiopathy-related inflammationJournal Article2016-08-0910.1016/j.ebcr.2016.05.003