What is DASH?

A central, open-access repository of research by members of the Harvard community.

Deposit Your Work

DASH enlarges the audience and impact of your work. Authors who deposit in DASH have access to on-demand metrics and receive monthly reports about their readership. Deposited works receive persistent URLs, are comprehensively indexed by search engines, including Google and Google Scholar, reach academic and non-academic readers who may not have access to the original publications, and are preserved by the Harvard Library.

Making your work open access in DASH is as simple as completing our quick submit form. We also welcome bulk deposits and offer CV scraping services. Simply contact OSC if you are interested. OSC will do the legal legwork for all submissions.

The OSC is pleased to offer a robust suite of services to support you and your scholarship. Visit our For Authors page to learn more.


  • This is How to Think about and Achieve Public Policy Success 

    Andrews, Matthew (Harvard Kennedy School, 2022-05)
    Officials working on public policies must answer questions like ‘What does policy success mean?’ and ‘How should I pursue policy work in order to achieve success?’ These are difficult questions, but there are ways to ...
  • Four Facts about Human Capital 

    Deming, David (Harvard Kennedy School, 2022-06)
    This paper synthesizes what economists have learned about human capital since Becker (1962) into four stylized facts. First, human capital explains at least one-third of the variation in labor earnings within countries and ...
  • The TORC1-Regulated CPA Complex Rewires an RNA Processing Network to Drive Autophagy and Metabolic Reprogramming 

    Tang, Hong-Wen; Hu, Yanhui; Chen, Chiao-Lin; Xia, Baolong; Zirin, Jonathan; Yuan, Min; Asara, John; Rabinow, Leonard; Perrimon, Norbert (Elsevier BV, 2018-05-01)
    Nutrient deprivation induces autophagy through inhibiting TORC1 activity. We describe a novel mechanism in Drosophila by which TORC1 regulates RNA processing of Atg transcripts and alters ATG protein levels and activities ...

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